Fichier de travail (INPUT) : ./DUMP-TEXT/1-11.txt
Encodage utilisé (INPUT) : UTF-8
Forme recherchée : obésité | obesity
_________________________________________________________________________________________________
- Ligne n°40 : Genes can push you toward obesity, but there are things you can do to prevent
Ligne n°41 : that ...- Ligne n°47 : The role of genes in obesity is one of science’s great puzzles, even
Ligne n°48 : though researchers have learned much in recent years. They have ...
Ligne n°53 : ... The experts are fairly confident about one thing, however: Except in- Ligne n°54 : rare instances, people genetically susceptible to obesity should not
Ligne n°55 : regard themselves as doomed. ...- Ligne n°57 : “I like to say that obesity is 80 percent genetic and 100 percent
Ligne n°58 : environmental,” says Philip F. Smith, co-director of the office of ...
Ligne n°58 : ... environmental,” says Philip F. Smith, co-director of the office of- Ligne n°59 : obesity research at the National Institute of Diabetes and Digestive
Ligne n°60 : and Kidney Diseases, or NIDDK. “You won’t become obese unless you ...
Ligne n°63 : ... A growing body of research suggests that although genes can tilt you- Ligne n°64 : toward obesity, you can prevent it with healthy behaviors, including
Ligne n°65 : diet and exercise. You might have to work a little harder, but it can ...
Ligne n°68 : ... “For most people, I can say unequivocally that genes are not your- Ligne n°69 : destiny,” Smith says. “They can predispose you to obesity, but only if
Ligne n°70 : you consume more calories than you burn off.” ...
Ligne n°73 : ... 17 percent of youths are obese, according to the Centers for Disease- Ligne n°74 : Control and Prevention. Worldwide, obesity has more than doubled since
Ligne n°75 : 1980, with more than 600 million adults afflicted, according to the ...
Ligne n°75 : ... 1980, with more than 600 million adults afflicted, according to the- Ligne n°76 : World Health Organization. The worldwide obesity rate among adults is
Ligne n°77 : 13 percent, far less than the U.S. rate. ...
Ligne n°80 : ... obese results in more deaths than being underweight, according to the- Ligne n°81 : WHO. Obesity is related to heart disease, stroke, Type 2 diabetes and
Ligne n°82 : certain types of cancer, including breast, colorectal and pancreatic. ...
Ligne n°82 : ... certain types of cancer, including breast, colorectal and pancreatic.- Ligne n°83 : According to a CDC estimate, the medical cost of obesity in the United
Ligne n°84 : States was $147 billion in 2008, the most recent year for which figures ...- Ligne n°87 : The quest to learn more about obesity genes began nearly 30 years ago
Ligne n°88 : with the launch of the first “twins” studies that compared siblings who ...
Ligne n°89 : ... were reared together with pairs who were reared apart. “They found that- Ligne n°90 : obesity had a strong genetic component,” says Marc Reitman, senior
Ligne n°91 : investigator and chief of NIDDK’s diabetes, endocrinology and obesity ...
Ligne n°90 : ... obesity had a strong genetic component,” says Marc Reitman, senior- Ligne n°91 : investigator and chief of NIDDK’s diabetes, endocrinology and obesity
Ligne n°92 : branch. ...
Ligne n°101 : ... identify about 200 loci, or genetic markers, pointing to genes that- Ligne n°102 : increase the risk of obesity. A genome-wide association study scans
Ligne n°103 : thousands of individuals’ complete sets of DNA looking for genetic ...
Ligne n°104 : ... changes that might be linked to a particular disease — in this case,- Ligne n°105 : obesity.
Ligne n°108 : ... road — are DNA regions probably located near genes responsible for- Ligne n°109 : obesity risk. These association studies suggest that obesity involves
- Ligne n°109 : obesity risk. These association studies suggest that obesity involves
Ligne n°110 : “not one gene, but a combination of many genes, and their influence ...
Ligne n°114 : ... Researchers using a genome-wide association study identified the first- Ligne n°115 : obesity-linked gene variants in 2007, related to what is known as the
Ligne n°116 : “fat mass and obesity-associated” (FTO) gene on chromosome 16. People ...
Ligne n°115 : ... obesity-linked gene variants in 2007, related to what is known as the- Ligne n°116 : “fat mass and obesity-associated” (FTO) gene on chromosome 16. People
Ligne n°117 : who carry an FTO variant have a 20 to 30 percent greater likelihood of ...- Ligne n°122 : “Because the genetic variants for obesity are numerous, it is virtually
Ligne n°123 : impossible for someone to not carry any of these variants,’’’ says ...
Ligne n°124 : ... Frank Hu, chair of the school’s department of nutrition, who has- Ligne n°125 : conducted a number of studies related to genes, obesity and diet.
Ligne n°126 : “However, some people may carry much fewer variants than others, but ...
Ligne n°133 : ... In a 2012 study, Hu and colleagues found that people genetically- Ligne n°134 : predisposed to obesity were more likely to experience increases in
Ligne n°135 : their BMI (body mass index, a measure of obesity) with a higher intake ...
Ligne n°134 : ... predisposed to obesity were more likely to experience increases in- Ligne n°135 : their BMI (body mass index, a measure of obesity) with a higher intake
Ligne n°136 : of beverages sweetened with sugar than their genetic peers who had ...
Ligne n°139 : ... Similarly, in a 2014 study, Hu and his team found that those with a- Ligne n°140 : genetic predisposition to obesity were more likely to gain weight if
Ligne n°141 : they ate fried foods four or five times a week than did those with the ...
Ligne n°149 : ... on a regular basis. Although we did not specifically analyze weight- Ligne n°150 : gain per se, the results on obesity imply that the findings would apply
Ligne n°151 : to weight gain as well.” ...
Ligne n°153 : ... Regularly consuming fried foods or sugary drinks “exacerbates the- Ligne n°154 : genetic effects of obesity,” Hu says. “Our findings indicate that
Ligne n°155 : genetic risk of obesity can be mitigated by simply changing an eating ...
Ligne n°154 : ... genetic effects of obesity,” Hu says. “Our findings indicate that- Ligne n°155 : genetic risk of obesity can be mitigated by simply changing an eating
Ligne n°156 : habit,” he says. ...
Ligne n°162 : ... with an FTO gene variant. Although those with the variant had a 23- Ligne n°163 : percent higher risk of obesity than those who did not have it, the
Ligne n°164 : researchers found that being physically active lowered the risk. In ...
Ligne n°164 : ... researchers found that being physically active lowered the risk. In- Ligne n°165 : fact, active adults with the gene variant showed a risk of obesity
Ligne n°166 : 27 percent lower than that of inactive adults. ...
Ligne n°168 : ... “As a hypothetical example, for a group of sedentary people who carry- Ligne n°169 : one FTO gene variant, their likelihood of obesity is 10 percent,” Hu
Ligne n°170 : says. “For a group of physically active people who also carry one gene ...
Ligne n°170 : ... says. “For a group of physically active people who also carry one gene- Ligne n°171 : variant, their likelihood of obesity would be lower, at 7.3 percent.”
Ligne n°173 : ... The results underscore “the importance of being physically active in- Ligne n°174 : obesity prevention, especially among those who are genetically
Ligne n°175 : predisposed to obesity,” Hu says. ...
Ligne n°174 : ... obesity prevention, especially among those who are genetically- Ligne n°175 : predisposed to obesity,” Hu says.
Ligne n°177 : ... The current body of research shows that “it absolutely is not a life- Ligne n°178 : sentence to obesity if you have one or more of these variants,” Smith
Ligne n°179 : says. ...- Ligne n°181 : The only exceptions to what is called “common obesity” — where many
Ligne n°182 : genes are involved in raising the risk of obesity — occur in what is ...
Ligne n°181 : ... The only exceptions to what is called “common obesity” — where many- Ligne n°182 : genes are involved in raising the risk of obesity — occur in what is
Ligne n°183 : known as “monogenic” obesity. This is when a mutation in a single gene ...
Ligne n°182 : ... genes are involved in raising the risk of obesity — occur in what is- Ligne n°183 : known as “monogenic” obesity. This is when a mutation in a single gene
Ligne n°184 : produces a very high risk of obesity, one that is difficult — sometimes ...
Ligne n°183 : ... known as “monogenic” obesity. This is when a mutation in a single gene- Ligne n°184 : produces a very high risk of obesity, one that is difficult — sometimes
Ligne n°185 : impossible — to counteract. But these cases are extremely rare. ...
Ligne n°187 : ... Still, researchers believe there is more to be done, including studies- Ligne n°188 : that look at whether any variants protect against obesity.
Ligne n°190 : ... “We haven’t thought enough about people who are resistant to the- Ligne n°191 : disease of obesity,” Smith says. “There are people out there who should
Ligne n°192 : be obese — look at all the bad food they eat and their lifestyle — and ...
Ligne n°202 : ... more,” Doumatey says. “The more you have of it, the better. Individuals- Ligne n°203 : who tend to have more adiponectin do not develop obesity.”
Ligne n°205 : ... Scientists also want to know more about the interplay between genes and- Ligne n°206 : the environment. Obesity clusters in families, but family members also
Ligne n°207 : tend to cook and eat the same foods and also may engage in the same ...
Ligne n°222 : ... “Why are some foods addictive? Why do we still eat when we feel full?”- Ligne n°223 : he adds. “The wiring and physiology around obesity is complicated, and
Ligne n°224 : we are just starting to try to figure it out.” ...